Who is more likely to develop diabetic ketoacidosis?
Diabetic ketoacidosis (DKA) is a life-threatening but preventable complication of diabetes characterized by uncontrolled hyperglycemia (>250 mg/dL), metabolic acidosis, and increased ketone concentration that occurs most frequently in persons with type 1 diabetes (2). CDC’s United States Diabetes Surveillance System* (USDSS) indicated an ...
What are the signs and dangers of diabetic ketoacidosis?
nausea and vomiting, abdominal pain and. dehydration. Risk factors for diabetic ketoacidosis are type 1 diabetes, and missing insulin doses frequently, or being exposed to a stressor requiring higher insulin doses (infection, etc).
Why is diabetic ketoacidosis so dangerous?
Diabetic ketoacidosis (DKA) is a serious problem that can happen in people with diabetes if their body starts to run out of insulin. When this happens, harmful substances called ketones build up in the body, which can be life-threatening if it's not found and treated quickly.
What causes ketoacidosis in people with diabetes?
According to the Mayo Clinic, the major symptoms of diabetic ketoacidosis include:
- Abdominal pain
- Confusion
- Excessive thirst
- Frequent urination
- Fruity-scented breath
- Loss of appetite
- Nausea and vomiting
- Shortness of breath
- Weakness or fatigue
Which disorder may result from DKA treatment?
Possible complications of the treatments Treatment complications include: Low blood sugar (hypoglycemia). Insulin allows sugar to enter your cells, causing your blood sugar level to drop. If your blood sugar level drops too quickly, you can develop low blood sugar.
Which medication is indicated for a patient with a pheochromocytoma quizlet?
Noncardioselective beta blockers, such as propranolol (Inderal) or nadolol (Corgard), are the usual choice; however, cardioselective agents, such as atenolol (Tenormin) and metoprolol (Lopressor), also may be used.
Which complication is associated with prolonged exposure to elevated glucocorticoids?
Although it may occur in children, Cushing syndrome most commonly affects adults between the ages of 25 to 40. It can be caused by prolonged exposure to elevated levels of glucocorticoids produced within the body (endogenous) or introduced from outside the body (exogenous).
Which intervention is indicated for treating central diabetes insipidus?
Central diabetes insipidus. Typically, this form is treated with a synthetic hormone called desmopressin (DDAVP, Nocdurna). This medication replaces the missing anti-diuretic hormone (ADH) and decreases urination. You can take desmopressin in a tablet, as a nasal spray or by injection.
Which disease is treated with Corticosteroidal hormone?
Medicine for Addison's disease Treatment usually involves corticosteroid (steroid) replacement therapy for life. Corticosteroid medicine is used to replace the hormones cortisol and aldosterone that your body no longer produces.
How are pheochromocytomas usually treated?
The primary treatment for a pheochromocytoma is surgery to remove the tumor. Before you have surgery, your health care provider will likely prescribe specific blood pressure medications. These medications block high-adrenaline hormones to lower the risk of developing dangerously high blood pressure during surgery.
What causes Cushing's syndrome?
Cushing's syndrome happens when you have too many corticosteroids in your body. When the disorder often starts with the pituitary gland, the condition is called Cushing's syndrome. The gland makes too much adrenocorticotropin hormone (ACTH). That causes the adrenal glands to make too many corticosteroids.
Which of the following is an effect of long-term glucocorticoid therapy?
Glucocorticoids induced Osteoporosis is one of the well-known and devastating adverse effects of long-term use of glucocorticoids. Up to 40% of patients on long-term glucocorticoids develop bone loss leading to fractures.
Which disease is caused by hypersecretion of glucocorticoids?
Hypersecretion of the glucocorticoid hormone cortisol leads to a disorder called Cushing's syndrome. The most common cause of Cushing's syndrome is a pituitary tumor, which causes excessive production of ACTH.
What does diabetes insipidus cause?
Diabetes insipidus is a rare disorder that causes the body to make too much urine. While most people make 1 to 3 quarts of urine a day, people with diabetes insipidus can make up to 20 quarts of urine a day. People with this disorder need to urinate frequently, called polyuria.
What disease is caused by deficiency of ADH?
Central diabetes insipidus (DI) is caused by a deficiency in antidiuretic hormone (ADH) and is characterized by polyuria in response to exogenous ADH analog2).
Can diabetes insipidus cause hyponatremia?
Conclusions: Hyponatremia is not commonly seen in patients with diabetes insipidus. However, it can occur if a patient presents with profound gastrointestinal losses or in adrenal crisis. Suspicion for diabetes insipidus should be raised when sodium level remains high despite free water replacement.
Diabetes Insipidus
What are the types of diabetes insipidus? Central Diabetes Insipidus The most common form of serious diabetes insipidus, central diabetes insipidus, results from damage to the pituitary gland, which disrupts the normal storage and release of ADH.
Diabetic Ketoacidosis (dka)
Sort What are symptoms and signs of Diabetic Ketoacidosis? 1. fatigue 2. tachypnea (kussmaul's respirations) 3. tachycardia 4. Altered mental status 5. abdominal pain 6. vomiting 7. polyuria 8. polydipsia What are potential precipitating causes of DKA? 1. recent or current infection of any type 2. Injury or trauma 3. ACS or MI 4. TIA or CVA 5.
Thyroid Disorders Case Study Quizlet
Some o sleep lisa hannigan sleep disorders psychology. # food for detox diet - weight loss detox center how to. The case keywords put sentences in order to make paragraph like insulin diabetes management ati quizlet did mess recently scientific study has found that your pituitary drives the thyroid.
Diabetes - Diabetic Ketoacidosis & Hyperosmolar Hyperglycemia Syndrome
Sort Hyperglycemia: Causes type of glucose level caused by 1) too much food, 2) too little diabetic medications, 3) inactivity, 4) emotional/physical stress, 5) poor absorption of insulin 6) illness 7) corticosteroids **counterregulatory hormones released when stress, illness persist Hyperglycemia: Manifestations manifests as 1) polyuria: osmotic diuresis (glucose in renal tubules cannot be reabsorbed; consequent hyperosmolarity and osmotic pressure results in more water in tubules) 2) polyphagia followed by lack of appetite, 3) polydipsia: hyperosmolarity of blood causes thirst as cells release more water into circulation 4) weakness/fatigue, 5) blurred vision, 6) glycosuria, 7) nausea/vomiting, 8) abdominal cramping 9) dry, warm, itchy skin Hyperglycemia: Treatment 1) exercise **do NOT exercise if BG 250 mg/dL (stress hormones released) and ketones (Type 1); do NOT exercise if >300 mg/dL (Type 2) 2) drink water 3) eat less CHO at meals **contact HCP if BG >250 mg/dL two-three times in one week During illness: 1) do NOT stop taking medication 2) check BG more frequently 3) clear liquids until no more nausea Hypoglycemia: Manifestations MILD: sweating, tremor, tachycardia, palpitation, nervousness, hunger MODERATE: poor concentration, numb lips/tongue, HA, light-headedness, slurred speech, irrational/combative behavior, visual disturbances SEVER: disorientation, loss of consciousness, difficult to arouse, seizures, coma **Can mimic alcohol intoxication.
Diabetes Mellitus
Sort Symptoms of Ketoacidosis Shortness of breath Stomach pain Fatigue Polydipsia, polyphagia Confusion Breath that smells fruity N/V A very dry mouth Muscle stiffness Symptoms of hypoglycemia Shakiness Dizziness Sweating Hunger Headache Pale skin color Sudden moodiness Clumsy or jerky movements Difficulty paying attention Insulin Regimens: 3 types *Fixed (standard/conventional) constant basal insulin rapid acting * Flexible (intensive) multiple daily injections of bolus insulin before meals Basal insulin 1-2 x/day * CSII-continuous subcutaneous insulin infusion a form of intensive therapy constant basal insulin bolus before meals - Dose based on body wt., then adjusted individually Continue reading >>.
Dka Vignette
Sort Describe the process of insulin release "1) Insulin is a hormone produced in the pancreatic β-cell and released in response to glucose influx into the β-cell cell through the GLUT-2 transporter 2) Glucose is then phosphorylated by glucokinase ultimately causing the ratio of ATP to ADP increases.
Like This Study Set?
Sort Ketogenesis FFA --> TG --> TCA cycle ---> ketones Ketones --> lungs ---> urine -->energy for brain, muscle, kidney Presenting complaint leth vom weakness sick if get to acidemic point if not sick but ketogenic...give insulin Diagnostics + why you would do each one minimum data - not standard but what you need to know to treat 1.